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Somatic mutations of the KEAP1 gene in common solid cancers
Nam Jin Yoo
, Hyung Ran Kim
, Yoo Ri Kim
, Chang Hyeok An
,
Sug Hyung Lee
Department of Pathology
The Catholic University of Korea
Research output
:
Contribution to journal
›
Article
›
peer-review
206
Scopus citations
Overview
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Biochemistry, Genetics and Molecular Biology
Solid
100%
Nested Gene
100%
Somatic Mutation
100%
KEAP1
100%
Mutation
54%
Transcription Factor Nrf2
36%
Protein
18%
Development
9%
Glutamine
9%
Cancer Cell
9%
Enzyme Active Site
9%
Single-Strand Conformation Polymorphism
9%
Cell Protection
9%
NAD(P)H Dehydrogenase (Quinone 1)
9%
Cysteine
9%
Ligase
9%
Medicine and Dentistry
Malignant Neoplasm
100%
Gene
100%
Somatic Mutation
100%
Lung
27%
Tumor Suppressor Protein
27%
Colon
18%
Breast
18%
Stomach
18%
Protein
18%
Development
9%
Analysis
9%
Leukemia
9%
Larynx
9%
Liver
9%
Carcinoma
9%
Cancer Cell
9%
Cancer Tissue
9%
Glutamine
9%
Gallbladder Cancer
9%
Cysteine
9%
Single Strand Conformation Polymorphism
9%
Cytoprotection
9%
Pharmacology, Toxicology and Pharmaceutical Science
Malignant Neoplasm
100%
Transcription Factor Nrf2
36%
Protein
18%
Leukemia
9%
Carcinoma
9%
Gallbladder Cancer
9%
Cysteine
9%
Reduced Nicotinamide Adenine Dinucleotide (Phosphate) Dehydrogenase (Quinone)
9%
Ligase
9%
Glutamine
9%
Cytoprotection
9%